Dear Johan,

I removed the link above and would appreciate your help and understanding regarding the following.

Could you please share the relevant content directly in the post rather than providing only a link to your website?

Articles published on this website should provide complete and meaningful information to readers. External links are welcome when they serve as references or additional resources, but they should not replace the main content of the article.

I have applied this policy to contributions from other authors as well, although I have been more flexible with your posts over the years. I would therefore appreciate your support in following the same approach going forward.

Thank you in advance for your understanding.

Kind regards,
Daniel

@daniel Hi Daniel, sure - I'll check how I can post it here. The reason I link to the blog is that these are long posts with tables, etc. I'm not selling anything 😉

@daniel, I converted the HTML to wpforo code and used the insert code option, left out links (references) but can't post because of the forum's settings: Forbidden. Message seems to be spam. Anti-Spam by CleanTalk.

Here's a summary instead:

The human gut produces ammonia through two main microbial processes: ureolysis, in which bacteria break down urea, and proteolytic deamination, in which bacteria ferment proteins and amino acids. Urease-producing bacteria are especially active in the mouth and stomach, while protein-fermenting bacteria generate most of the ammonia found in the distal colon.

The clearest connection between microbial ammonia and cancer involves Helicobacter pylori. This bacterium uses urease to survive in the stomach, producing ammonia that can damage the stomach lining, weaken protective barriers and encourage abnormal cell growth. H. pylori is an established cause of gastric cancer and MALT lymphoma.

In the colon, several ammonia-producing or protein-fermenting bacteria are associated with colorectal cancer. Some promote cancer mainly through toxins, DNA-damaging compounds, inflammation or immune evasion rather than through ammonia alone. These include certain strains of Morganella morganii, Bacteroides fragilis, Fusobacterium nucleatum and Klebsiella pneumoniae.

Research also suggests that ammonia itself may interfere with TGF-beta, an important tumour-suppressing pathway. In animal experiments involving a high-fat diet, ammonia-producing Bacteroides species disrupted this protective signalling while promoting inflammation. This provides a possible connection between diet, microbiome imbalance, ammonia production and colorectal cancer.

These effects are highly dependent on the bacterial strain, diet and surrounding microbial community. Some species associated with harmful ammonia production can also have beneficial effects under different conditions. Clostridium perfringens, for example, appears more often to be a marker of an existing gastrointestinal cancer than a direct cause, while one of its toxins is also being investigated as a potential cancer treatment.
Overall, ammonia appears to be one contributor within a much larger network of microbial metabolites, toxins and inflammatory processes. The strongest direct evidence concerns H. pylori in the stomach, while much of the evidence for colonic bacteria still comes from animal and laboratory studies. Finding one of these bacteria does not by itself indicate that a person has cancer or will develop it.

@j Hi Johan,

Thank you for understanding.

I know you are not selling anything, and that was not my concern. The purpose of this website has always been to make useful and meaningful information directly available to readers, and I would like to preserve that principle.

For very long articles or posts containing extensive tables, a concise summary with the main information would be perfectly fine and elegant, together with a link to the complete article on your blog.

Thank you again for your help with this.

Kind regards,
Daniel